Gut and Liver is an international journal of gastroenterology, focusing on the gastrointestinal tract, liver, biliary tree, pancreas, motility, and neurogastroenterology. Gut atnd Liver delivers up-to-date, authoritative papers on both clinical and research-based topics in gastroenterology. The Journal publishes original articles, case reports, brief communications, letters to the editor and invited review articles in the field of gastroenterology. The Journal is operated by internationally renowned editorial boards and designed to provide a global opportunity to promote academic developments in the field of gastroenterology and hepatology. +MORE
Yong Chan Lee |
Professor of Medicine Director, Gastrointestinal Research Laboratory Veterans Affairs Medical Center, Univ. California San Francisco San Francisco, USA |
Jong Pil Im | Seoul National University College of Medicine, Seoul, Korea |
Robert S. Bresalier | University of Texas M. D. Anderson Cancer Center, Houston, USA |
Steven H. Itzkowitz | Mount Sinai Medical Center, NY, USA |
All papers submitted to Gut and Liver are reviewed by the editorial team before being sent out for an external peer review to rule out papers that have low priority, insufficient originality, scientific flaws, or the absence of a message of importance to the readers of the Journal. A decision about these papers will usually be made within two or three weeks.
The remaining articles are usually sent to two reviewers. It would be very helpful if you could suggest a selection of reviewers and include their contact details. We may not always use the reviewers you recommend, but suggesting reviewers will make our reviewer database much richer; in the end, everyone will benefit. We reserve the right to return manuscripts in which no reviewers are suggested.
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Moon Kyung Joo, Jong-Jae Park*, Beom Jae Lee, Ji Hoon Kim, Jong Eun Yeon, Jae Seon Kim, Kwan Soo Byun, and Young-Tae Bak
Department of Internal Medicine, Korea University College of Medicine, Seoul, Korea.
Correspondence to: Jong-Jae Park. Department of Internal Medicine, Korea University Guro Hospital, Korea University College of Medicine, 97 Gurodong-gil, Guro-dong, Guro-gu, Seoul 152-703, Korea. Tel: +82-2-2626-3003, Fax: +82-2-854-8453, gi7pjj@yahoo.co.kr
This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
Gut Liver 2011;5(2):238-241. https://doi.org/10.5009/gnl.2011.5.2.238
Published online June 28, 2011, Published date June 30, 2011
Copyright © Gut and Liver.
Invasive gastric
Keywords: Candidiasis, Stenosis, Self-expandable metallic stent
Normally colonized
A 69-year-old man with type 2 diabetes mellitus was referred to Division of Gastroenterology, Korea University Guro Hospital with anorexia, nausea, vomiting, and epigastric pain. He was diagnosed as diabetes 15 years ago and his blood sugar was controlled by oral hypoglycemic agents. He had undergone subtotal gastrectomy with Billroth-I anastomosis due to advanced gastric cancer (Borrman type 3, T2N0M0) before one month from referral. He had suffered from drowsiness and purulent sputum, and was treated with intravenous antibiotics due to aspiration pneumonia during post-operative care. He did not receive any anti-acid agents, anti-H-2 receptors or oral proton pump inhibitors (PPIs) at that time. His absolute neutrophil count was within normal limit and anti-human immunodeficiency virus (HIV) antibody was negative. An esophagogastroduodenoscopy showed a diffuse mucosal defect at remnant stomach body, which was covered with greenish to yellowish plaque and exudates (Fig. 1). Biopsies were performed at ulcerative lesions and its histologic findings demonstrated that there were many yeast forms of fungal organism with chronic active ulcer, which was compatible with gastric Candidiasis (Fig. 2). Oral fluconazole was administered for more than two weeks, however follow-up esophagogastroduodenoscopy could not show any improvement of above mentioned lesion. Therefore, amphoterecin B was given intravenously for 10 days. His symptoms and endoscopic findings were improved and he was discharged after completion of intravenous amphoterecin B treatment.
However, his nausea and vomiting recurred after discharge, thus he underwent follow-up esophagogastroduodenoscopy. Ulcerative lesion was much improved comparing with previous findings, however stenotic change at pre-anastomosis site was developed and tip of the scope could not be passed through the narrowing portion (Fig. 3A). Gastroduodenography also indicated partial narrowing near anastomosis site (Fig. 3B). Therefore we inserted SEMS (Bonastent®, covered; Standard Sci Tech, Seoul, Korea) through the anastomosis site at three months after referral (Fig. 4A). After the procedure, his symptom was nearly resolved and did not recur thereafter. On follow-up esophagogastroduodenoscopy which was performed at two months after procedure, the stent was migrated from the anastomosis site and expelled outside the gastrointestinal tract spontaneously. However, anastomosis site remained dilated and the tip of endoscope could be passed through well (Fig. 4B).
There are many conditions which are known to contribute to the colonization of
Endoscopically, gastric
As we mentioned above, stenosis can be developed if diffuse
As far we know, there has been no reported case of stenotic change after fungal infection and its management with temporary stent insertion. We report a case of successful placement of SEMS in patient who had subtotal gastrectomy and stenotic change at anastomosis site after diffuse invasive gastric
Gut Liver 2011; 5(2): 238-241
Published online June 30, 2011 https://doi.org/10.5009/gnl.2011.5.2.238
Copyright © Gut and Liver.
Moon Kyung Joo, Jong-Jae Park*, Beom Jae Lee, Ji Hoon Kim, Jong Eun Yeon, Jae Seon Kim, Kwan Soo Byun, and Young-Tae Bak
Department of Internal Medicine, Korea University College of Medicine, Seoul, Korea.
Correspondence to: Jong-Jae Park. Department of Internal Medicine, Korea University Guro Hospital, Korea University College of Medicine, 97 Gurodong-gil, Guro-dong, Guro-gu, Seoul 152-703, Korea. Tel: +82-2-2626-3003, Fax: +82-2-854-8453, gi7pjj@yahoo.co.kr
This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
Invasive gastric
Keywords: Candidiasis, Stenosis, Self-expandable metallic stent
Normally colonized
A 69-year-old man with type 2 diabetes mellitus was referred to Division of Gastroenterology, Korea University Guro Hospital with anorexia, nausea, vomiting, and epigastric pain. He was diagnosed as diabetes 15 years ago and his blood sugar was controlled by oral hypoglycemic agents. He had undergone subtotal gastrectomy with Billroth-I anastomosis due to advanced gastric cancer (Borrman type 3, T2N0M0) before one month from referral. He had suffered from drowsiness and purulent sputum, and was treated with intravenous antibiotics due to aspiration pneumonia during post-operative care. He did not receive any anti-acid agents, anti-H-2 receptors or oral proton pump inhibitors (PPIs) at that time. His absolute neutrophil count was within normal limit and anti-human immunodeficiency virus (HIV) antibody was negative. An esophagogastroduodenoscopy showed a diffuse mucosal defect at remnant stomach body, which was covered with greenish to yellowish plaque and exudates (Fig. 1). Biopsies were performed at ulcerative lesions and its histologic findings demonstrated that there were many yeast forms of fungal organism with chronic active ulcer, which was compatible with gastric Candidiasis (Fig. 2). Oral fluconazole was administered for more than two weeks, however follow-up esophagogastroduodenoscopy could not show any improvement of above mentioned lesion. Therefore, amphoterecin B was given intravenously for 10 days. His symptoms and endoscopic findings were improved and he was discharged after completion of intravenous amphoterecin B treatment.
However, his nausea and vomiting recurred after discharge, thus he underwent follow-up esophagogastroduodenoscopy. Ulcerative lesion was much improved comparing with previous findings, however stenotic change at pre-anastomosis site was developed and tip of the scope could not be passed through the narrowing portion (Fig. 3A). Gastroduodenography also indicated partial narrowing near anastomosis site (Fig. 3B). Therefore we inserted SEMS (Bonastent®, covered; Standard Sci Tech, Seoul, Korea) through the anastomosis site at three months after referral (Fig. 4A). After the procedure, his symptom was nearly resolved and did not recur thereafter. On follow-up esophagogastroduodenoscopy which was performed at two months after procedure, the stent was migrated from the anastomosis site and expelled outside the gastrointestinal tract spontaneously. However, anastomosis site remained dilated and the tip of endoscope could be passed through well (Fig. 4B).
There are many conditions which are known to contribute to the colonization of
Endoscopically, gastric
As we mentioned above, stenosis can be developed if diffuse
As far we know, there has been no reported case of stenotic change after fungal infection and its management with temporary stent insertion. We report a case of successful placement of SEMS in patient who had subtotal gastrectomy and stenotic change at anastomosis site after diffuse invasive gastric